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Brain 2006 129(4):830-831; doi:10.1093/brain/awl035
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© The Author (2006). Published by Oxford University Press on behalf of the Guarantors of Brain. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Scientific Commentary

Frontal temporal dementia: dissecting the aetiology and pathogenesis

The first 10% of the full text of this article appears below.

Satisfactory classification of frontotemporal dementias (FTD) has always been difficult with historical schemes being based on clinical symptoms and on pathology (reviewed, Kertesz, 2005Go) and more recent proposals focused on genetics (Foster et al., 1997Go). Concordance between these schemes is imperfect and the situation further complicated by the growing realization that there is a clinical, pathological and aetiological overlap between amyotrophic lateral sclerosis (ALS) and FTD.

A milestone in our understanding of FTD occurred at a consensus conference where genetics groups pooled their data and realized that a large proportion of FTD families showed linkage to chromosome 17 around the tau (MAPT) locus . . . [Full Text of this Article]

John Hardy1, Parastoo Momeni1 and Bryan J. Traynor2

1 Laboratory of Neurogenetics, National Institute on Aging and 2 Section on Developmental and Genetic Epidemiology, National Institute of Mental Health, Porter Neuroscience Building, 35 Convent Drive, Bethesda, MD, USA E-mail: hardyj@mail.nih.gov


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Home page
 Hum Mol GenetHome page
K. Talbot and O. Ansorge
Recent advances in the genetics of amyotrophic lateral sclerosis and frontotemporal dementia: common pathways in neurodegenerative disease
Hum. Mol. Genet., October 15, 2006; 15(suppl_2): R182 - R187.
[Abstract] [Full Text] [PDF]