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Brain Advance Access published online on February 19, 2008
Brain, doi:10.1093/brain/awn027
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© The Author (2008). Published by Oxford University Press on behalf of the Guarantors of Brain. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Does CNTF mediate the effect of intraocular inflammation on optic nerve regeneration?

Qi Cui1, Larry Benowitz2 and Yuqin Yin2

1The Chinese University of Hong Kong, Hong Kong, China and 2Children's Hospital and Harvard Medical School, Boston, MA, USA

Correspondence to: L. Benowitz, 300 Longwood Avenue, Boston, MA 02115, USA E-mail: larry.benowitz@childrens.harvard.edu

Received December 27, 2007. Accepted January 27, 2008.

The first 10% of the full text of this article appears below.

Sir, we wish to comment on the paper ‘Astrocyte-derived CNTF switches mature retinal ganglion cells (RGCs) to a regenerative state following inflammatory stimulation’ (Muller et al., 2007Go). RGCs, the projection neurons of the eye, are normally unable to regenerate their axons if the optic nerve is injured. However, they can be stimulated to do so by inducing an inflammatory reaction in the eye (Leon et al., 2000Go; Yin et al., . . . [Full Text of this Article]


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