Skip Navigation

This Article
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (24)
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by BLUME, W. T.
Right arrow Articles by MCLACHLAN, R. S.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by BLUME, W. T.
Right arrow Articles by MCLACHLAN, R. S.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Brain, Vol. 115, No. 5, 1509-1520, 1992
© 1992 Guarantors of Brain


research-article

SEIZURES INVOLVING SECONDARY SENSORY AND RELATED AREAS

WARREN T. BLUME, DANIEL C. JONES, G. BRYAN YOUNG, JOHN P. GRAVIN and RICHARD S. MCLACHLAN

University Hospital, The University of Western Ontario London, Ontario, Canada

Correspondence to: Correspondence to Dr W. T. Blume, University Hospital, 339 Windermere Road, London, Ontario, Canada, N6A 5A5.

Five patients with seizures involving the secondary sensory and/or related areas (SSRA) are presented. Four of five experienced ictal numbness and'or tingling bilaterally and/or axially, this involved fingertips (three patients), lips (two), tongue (two), and was diffuse in one. The fifth patient experienced bilateral ictal pain. Associated ictal symptoms implicating adjacent regions appeared in all five patients, including contralateral clonic movements (two patients), hypersalivation (two), taste (one), vocalization (two), dysphagia (one), and contralateral sensory march (one). Two patients had ictal symptoms suggestive of adjacent temporal lobe involvement. By history, the SSRA was involved at seizure onset in four and by spread in one. All five patients had electroencephalogram (EEG) or subdural EEG supportive evidence of SSRA involvement: ictal (three) and interictal (three). Three patients had lesions in this area shown by magnetic resonance imaging or computerized tomography and all three had histologically proven glial tumours. Relevant experimental physiological and anatomical data are reviewed.

Received September 23, 1991. Revised April 22, 1992. Accepted April 30, 1992.


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
Anesth. Analg.Home page
T. Sprenger, M. Valet, R. Woltmann, C. Zimmer, R. Freynhagen, E. F. Kochs, T. R. Tolle, and K. J. Wagner
Imaging pain modulation by subanesthetic s-(+)-ketamine.
Anesth. Analg., September 1, 2006; 103(3): 729 - 737.
[Abstract] [Full Text] [PDF]


Home page
J. Neurol. Neurosurg. PsychiatryHome page
J Yamamoto, A Ikeda, M Matsuhashi, T Satow, M Takayama, S Ohara, R Matsumoto, N Mikuni, J Takahashi, S Miyamoto, et al.
Seizures arising from the inferior parietal lobule can show ictal semiology of the second sensory seizure (SII seizure)
J. Neurol. Neurosurg. Psychiatry, March 1, 2003; 74(3): 367 - 369.
[Abstract] [Full Text] [PDF]



Disclaimer: Please note that abstracts for content published before 1996 were created through digital scanning and may therefore not exactly replicate the text of the original print issues. All efforts have been made to ensure accuracy, but the Publisher will not be held responsible for any remaining inaccuracies. If you require any further clarification, please contact our Customer Services Department.