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Brain 2006 129(1):E39; doi:10.1093/brain/awh697
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© The Author (2005). Published by Oxford University Press on behalf of the Guarantors of Brain. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Letter to the Editor

Silent or non-clinical infarct-like lesions in the posterior circulation territory in migraine: brain hypoperfusion or hyperperfusion?

Vinod Gupta

Dubai Police Medical Services, PO Box 12005, Dubai, United Arab Emirates

E-mail: docgupta@emirates.net.ae

Received October 7, 2005. Revised October 20, 2005. Accepted October 24, 2005.

The first 10% of the full text of this article appears below.

Kruit et al. (2005)Go described infratentorial predominantly cerebellar silent or non-clinical infarct-like lesions in a cohort of migraine patients with or without aura; these authors concluded that such discrete lesions represent the effects of episodic focal brain hypoperfusion coupled to embolic damage and elaborated upon a specific pathophysiological role for the cerebellum. While Kruit et al. (2005)Go attributed brain hypoperfusion to cortical spreading depression (CSD) there is a significant unbridged conceptual gap between CSD and cerebral oligaemia (Pearce, 1985Go; Blau, 1992Go. . . [Full Text of this Article]


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